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EDITORIAL BOARD
 
  Dr. VIJAY MENON  
 
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Title : Dr.
First Name : VIJAY
Last Name : MENON
University/Institution : Icahn School of Medicine at Mount Sinai
Phone # : 804-938-0084
Email ID : vijay.menon@mssm.edu
City : New York
Country : United States
State : New york
Zipcode : 10029
Department : Cell, Developmental, and Regenerative Biology
Company Name :
Area of Research
Erythroid Cell Biology and Mitochondrial Biology
Area of Expertise
Cancer Biology, DNA damage response and repair, mTOR signaling, Cellular and Molecular Biology
Brief Description of Research Interest :
My research interests essentially fall under DNA Damage Response (DDR) and Repair and Erythroid Biology. 
My earlier work has been on platinum based compounds and their mechanism of action in cancer cells. I then moved on to study the role of cell cycle related kinases in DDR and mTOR signaling. Currently, I am working toward understanding the mechanism of mitochondrial regulation during erythropoiesis using mouse models.
Representative Publications :

Menon V and Ghaffari S. Transcription Factors FOXO in the Regulation of Homeostatic Hematopoiesis. Current Opinion in Hematology. 2018 (Invited Review).

Menon V and Povirk L. XLF/Cernunnos: An important but puzzling participant in the nonhomologous end joining DNA repair pathway. DNA Repair. 2017

Menon V and Povirk L. End-processing nucleases and phosphodiesterases: An elite supporting cast for the non-homologous end joining pathway of DNA double-strand break repair. DNA Repair. 2016

PetersonEJ, Menon VR, Gatti L,Kipping R, Dewasinghe D, Perego P, Povirk LF, Farrell N. Nucleolar Targeting by Platinum. p53-independent Apoptosis Follows rRNA Inhibition, Cell-cycle Arrest and DNA Compaction. MolecularPharmaceutics. 2014

Menon V,Peterson E, Valerie K, Farrell N, Povirk L. Ligand modulation of a dinuclear platinum compound leads to mechanistic differences in cell cycle progression and arrest. Biochemical Pharmacology.2013

Akopiants K, Mohapatra S, Menon V, ZhouT, Valerie K, Povirk L. Tracking the processing of damaged DNA double-strand break ends by ligation-mediated PCR: Increased persistence of3´-phosphoglycolate termini in SCAN1 cells. NucleicAcids Research. 2013

Mohapatra S, Yannone S, Lee Suk-Hee,  Hromas R,Akopiants K, Menon V, Ramsden D, Povirk L. Trimming of damaged 3′ overhangs of DNA double-strand breaks bythe Metnase and Artemis endonucleases. DNARepair (Amst. 2013)


 
     
 
 
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